[1] GENG T Y,XIA L L,LI F Y,et al.The role of endoplasmic reticulum stress and insulin resistance in the occurrence of goose fatty liver[J].Biochem Biophys Res Commun,2015,465(1):83-87.
[2] GENG T Y,YANG B,LI F Y,et al.Identification of protective components that prevent the exacerbation of goose fatty liver:Characterization,expression and regulation of adiponectin receptors[J].Comp Biochem Physiol B Biochem Mol Biol,2016,194-195:32-38.
[3] LIU L,ZHAO X,WANG Q,et al.Prosteatotic and protective components in a unique model of fatty liver:Gut microbiota and suppressed complement system[J].Sci Rep,2016,6:31763.
[4] OSMAN R H,SHAO D,LIU L,et al.Expression of mitochondria-related genes is elevated in overfeeding-induced goose fatty liver[J].Comp Biochem Physiol B Biochem Mol Biol,2016,192:30-37.
[5] GENG T,HU W,BROADWATER M H,et al.Fatty acids differentially regulate insulin resistance through endoplasm reticulum stress-mediated induction of tribbles homologue 3:a potential link between dietary fat composition and the pathophysiological outcomes of obesity[J].Diabetologia,2013,56(9):2078-2087.
[6] LEE A S.The glucose-regulated proteins:stress induction and clinical applications[J].Trends Biochem Sci,2001,26(8):504-510.
[7] TABAS I,RON D.Integrating the mechanisms of apoptosis induced by endoplasmic reticulum stress[J].Nat Cell Biol,2011,13(3):184-190.
[8] RAO R V,ELLERBY H M,BREDESEN D E.Coupling endoplasmic reticulum stress to the cell death program[J].Cell Death Differentiation,2004,11(4):372-380.
[9] SIDRAUSKI C,WALTER P.The transmembrane kinase Ire1p is a site-specific endonuclease that initiates mRNA splicing in the unfolded protein response[J].Cell,1997,90(6):1031-1039.
[10] RON D,WALTER P.Signal integration in the endoplasmic reticulum unfolded protein response[J].Nat Rev Mol Cell Biol,2007, 8(7):519-529.
[11] DENG J,LU P D,ZHANG Y H,et al.Translational repression mediates activation of nuclear factor kappa B by phosphorylated translation initiation factor 2[J].Mol Cell Biol,2004,24(23):10161-10168.
[12] HU P,HAN Z,COUVILLON A D,et al.Autocrine tumor necrosis factor alpha links endoplasmic reticulum stress to the membrane death receptor pathway through IRE1α-mediated NF-κB activation and down-regulation of TRAF2 expression[J]. Mol Cell Biol,2006,26(8):3071-3084.
[13] CULLINAN S B,DIEHL J A.Coordination of ER and oxidative stress signaling:the PERK/Nrf2 signaling pathway[J].Int J Biochem Cell Biol,2006,38(3):317-332.
[14] GOTOH T,MORI M.Nitric oxide and endoplasmic reticulum stress[J].Arterioscler Thromb Vasc Biol,2006,26(7):1439-1446.
[15] JI C,KAPLOWITZ N,LAU M Y,et al.Liver-specific loss of glucose-regulated protein 78 perturbs the unfolded protein response and exacerbates a spectrum of liver diseases in mice[J].Hepatology,2011,54(1):229-239.
[16] TEODORO-MORRISON T,SCHUIKI I,ZHANG L,et al.GRP78 overproduction in pancreatic beta cells protects against high-fat-diet-induced diabetes in mice[J].Diabetologia,2013,56(5):1057-1067.
[17] PALLEPATI P,AVERILL-BATES D A.Mild thermotolerance induced at 40℃ protects HeLa cells against activation of death receptor-mediated apoptosis by hydrogen peroxide[J].Free Radic Biol Med,2011,50(6):667-679.
[18] SAGE A T,HOLTBY-OTTENHOF S,SHI Y Y,et al.Metabolic syndrome and acute hyperglycemia are associated with endoplasmic reticulum stress in human mononuclear cells[J].Obesity,2012,20(4):748-755.
[19] LUO S Z,MAO C H,LEE B,et al.GRP78/BiP is required for cell proliferation and protecting the inner cell mass from apoptosis during early mouse embryonic development[J].Mol Cell Biol,2006,26(15):5688-5697.
[20] KONG L N,ZHANG D X,JI C L,et al.Association analysis between SNPs in the 5'-flanking region of the chicken GRP78 gene,thermotolerance parameters,and tissue mRNA expression[J].Genet Mol Res,2015,14(2):6110-6123.
[21] OSMAN R H,LIU L,XIA L L,et al.Fads1 and 2 are promoted to meet instant need for long-chain polyunsaturated fatty acids in goose fatty liver[J].Mol Cell Biochem,2016,418(1-2):103-117.
[22] EKSTEDT M,HAGSTRÖM H,NASR P,et al.Fibrosis stage is the strongest predictor for disease-specific mortality in NAFLD after up to 33 years of follow-up[J].Hepatology,2015,61(5):1547-1554.
[23] DU K Y,HERZIG S,KULKARNI R N,et al.TRB3:a tribbles homolog that inhibits Akt/PKB activation by insulin in liver[J].Science,2003,300(5625):1574-1577.
[24] GENG T Y,XING Z,XIA L L,et al.Supplementing dietary sugar promotes endoplasmic reticulum stress-independent insulin resistance and fatty liver in goose[J].Biochem Biophys Res Commun,2016,476(4):665-669.
[25] LAI S J,LI Y,KUANG Y,et al.PKCδ silencing alleviates saturated fatty acid induced ER stress by enhancing SERCA activity[J].Biosci Rep,2017,37(6):BSR20170869.
[26] DING S T,LIU B H,KO Y H.Cloning and expression of porcine adiponectin and adiponectin receptor 1 and 2 genes in pigs[J].J Anim Sci,2004,82(11):3162-3174.
[27] WANG Y J,WANG X L,LAU W B,et al.Adiponectin Inhibits TNF-α-induced vascular inflammatory response via caveolin-mediated ceramidase recruitment and activation[J].Circ Res,2014,114(5):792-805.
[28] MACEYKA M,SPIEGEL S.Sphingolipid metabolites in inflammatory disease[J].Nature,2014,510(7503):58-67.
[29] LI T,JIANG S,LU C,et al.Snapshots:Endoplasmic reticulum stress in lipid metabolism and cardiovascular disease[J].Curr Issues Mol Biol,2018,28:14-28.
[30] JING L,JIA X W.Lycium barbarum polysaccharide arbitrates palmitate-induced apoptosis in MC3T3-E1 cells through decreasing the activation of ERS-mediated apoptosis pathway[J].Mol Med Rep,2018,17(2):2415-2421.
[31] WANG L L,HU R C,DAI A G,et al.Bevacizumab induces A549 cell apoptosis through the mechanism of endoplasmic reticulum stress in vitro[J].Int J Clin Exp Pathol,2015,8(5):5291-5299. |